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98 ; Abramson M, Littlejohn GO. Hepatic reactions to nifedipine. Med J Aust 1985; 142 1 ; : 47-48. 99 ; Biour M, Grange JD, Barbare JC, Legendre C, Geri D, Valty J et al. Atteinte hpatique due la nifedipine. Description d'un cas et revue de la littrature. Therapie 1987; 42 3 ; : 301-303. 100 ; Davidson AR. Lymphocyte sensitisation in nifedipine-induced hepatitis. Br Med J 1980; 281 6251 ; : 1354. 101 ; Kiire CF, Rutherford D. Nifedipine-associated jaundice: a second case. East Afr Med J 1986; 63 8 ; : 560-561. 102 ; Rotmensch HH, Roth A, Liron M, Rubinstein A, Gefel A, Livni E. Lymphocyte sensitisation in nifedipine-induced hepatitis. Br Med J 1980; 281 6246 ; : 976-977. 103 ; Shaw DR, Misan GM, Johnson RD. Nifedupine hepatitis. Aust N Z J Med 1987; 17 4 ; : 447-448. 104 ; Welch HG, Lazar B, Gresser J, McMahon BJ. Nifedipine-induced hepatitis. Alaska Med 1986; 28 1 ; : 11-12. 105 ; Benichou C. Criteria of drug-induced liver disorders. Report of an international consensus meeting. J Hepatol 1990; 11 2 ; : 272-276. 106 ; Lang I, Huber K, Capek J, Glogar DH, Probst P, Kaindl F. Furosemide and increases in liver enzymes. Ann Intern Med 1988; 109 10 ; : 845. 107 ; Bellido Casado J, Fernando de Frutos Arribas J, del Rio Fernandez MdC, Mena Martin J. Hepatitis y furosemida. Rev Esp Enferm Dig 1996; 88 11 ; : 813-814. 108 ; Lagarriga J, Buenrrostro C, Rodriguez P, Castaneda J. Hepatonecrosis por furosemida. Lesion privativa de algunas especies? Rev Gastroenterol Mex 1977; 42 3 ; : 117-125.
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Elliott HM. PA. Clinical pharmacokinetics of nifedipine. Implications for the care of the elderly. Drugs and Aging 1997: 1 Lobo JJ. DB: Kendall MJ. The intra- and inter-subject variability of nifedipine pharmacokinetics in young volunteers. European Journal of Clinical Pharmacology 1986. Traditional plan designs attempt to maintain a balance between the costs that the plan incurs and the share it asks its members to contribute. A common cost-sharing strategy is to require either a co-payment or coinsurance amount for prescription purchases. A co-payment is a fixed-dollar amount, whereas coinsurance is a percentage of a medication's cost. The typical co-payment and coinsurance amounts required by Medco clients are summarized in Tables 1 and 2. Table 1. Drug Allergy and Drug Interaction Alerts in Primary Care, by Physicians' Decisions to Override, because nifedipine 10 mg.
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Found to be well correlated with the binding constants 2410 4 , 16510 4 and 14104 ; for the interaction nifedipine, verapamil and PGF2 with DPPC liposomes measured earlier Purnima and Kothekar, 1988a ; . It had been suggested by Shi and Tien 1986 ; that fluidity effect of different drugs on membrane lipids depend on their lipophilicity. In the theory proposed by Lee 1976 ; for sodium channels, fluidity of the lipid moiety leads to closing of the channels. Whether a similar model can be used to explain the pharmacological function of Ca2 + mediators is not known. The greater effect of nifedipine is indicative of the fact that the theory may hold true for calcium mediators also. In our study PGF2 shows the least effect and it is an agonist. Thus, effect on fluidity of lipid membranes is a useful indicator of the differential activity of Ca2 + mediators. However, we cannot overlook the fact that the physiological mechanism for Ca2 + transport is more complex and involves many parameters, such as state of membrane polarization, ion fluxes, extracellular calcium levels, etc. Akiyama and Gish, 1979; Herbette et al., 1983; Reddy et al., 1984; Kothekar et al., 1985 ; . We have studied the influence of calcium ion on lipid fluidity of D P and D M P vesicles in the presence of 3 drugs to probe further into the interrelationship between various factors controlling Ca2 + transport. Effect of Ca2 + mediators on membrane fluidity in the presence of Ca2 + ions Figure 4a shows that addition of Ca2 + ion 054 mM ; to DPPC liposomes increases fluorescence polarization, which means that it causes stiffening of the membrane. This is due to increase in the lipid order parameter S, which shifts the transition temperature to the higher side Trauble and Eibl, 1974; Eibl and Blume, 1979 ; . Decrease in probe mobility was also noticed by Ashley and Brammer 1984 ; upon addition of CaCl2 to synaptosomal lipid extracts. Addition of nifedipine and verapamil lead to further increase of and increase in the transition temperature by 7 and 3C respectively. Contrary to this, addition of PGF2 leads to decrease of and reduction of Tc by. 2C which means that there is fluidization of the membrane like in the case of action of anaesthetics observed by Papahadjopoulos et al 1975 ; . This result shows the complexity of the phenomenon and the need for a detailed study of the interaction between channel-forming proteins and lipids in the presence of drugs and other molecules. In the case of DMPC liposomes figure 4b ; , the presence of Ca2 + leads to increase in and disappearance of gel-liquid-crystalline transition. Addition, of nifedipine, verapamil and PGF2 leads to a complicated behaviour. Transition temperature decreases by 2C upon addition of nifedipine. Verapamil and PGF2 cause a reduction in Tc by 15C. At any temperature PGF2 brings about the maximum fluidization. Conclusions The results show that there are differences in the effect on membrane fluidity of different Ca2 + mediators. The effect depends on the nature of fatty acid chains attached to the lipid molecules and the environment of the molecules. It is difficult to ascertain whether quantitative differences in fluidity changes are significant for differential activity of Ca2 + mediators or whether differences in the.
George et al. PharmacoEconomics 2001; 19 11 ; : 1103-1109 and selegiline, for example, nifedipine er drug. Table 3. Effects of different doses of compound 6a-6i ; on PTZ-induced seizures in mice. Compound Dose Seizure latency Seizure duration Mortality % 5 57.15 6a Niffedipine 2.710.52 25.003.70 10 Nifedipin3 3.270.42 24.283.19 71.43 Control 9.002.60 49.579.82 Intraperitoneal injecation of the compounds 30 minutes before PTZ decreased seizure duration significantly but did not show any significant effects on seizure latency and mortrality. Each point show mean S. E M animals. * p 0.05 showed significant difference statistically. 3 amiodarone hcl tablet amiodarone hcl vial CARDENE I.V. AMPUL CARDIZEM DISP SYRIN CORDARONE I.V. AMPUL CORDARONE TABLET digoxin ampul digoxin solution digoxin tablet diltiazem hcl cap. sr 12h diltiazem hcl cap. sr 24h diltiazem hcl capsule cr diltiazem hcl capsule sa diltiazem hcl tablet diltiazem hcl vial disopyramide phosphate capsule disopyramide phosphate capsule sa felodipine tab. sr 24h flecainide acetate tablet isosorbide dinitrate tab subl isosorbide dinitrate tablet isosorbide dinitrate tablet sa isosorbide mononitrate tab. sr 24h isosorbide mononitrate tablet isradipine capsule LANOXICAPS CAPSULE LANOXIN AMPUL LANOXIN PEDIATRIC AMPUL LANOXIN TABLET lidocaine hcl pf syringe mexiletine hcl capsule nicardipine hcl capsule nifedipine capsule nifedipine tab nifedipine tablet sa nitroglycerin capsule sa nitroglycerin patch td 24 nitroglycerin tab subl 1 3 NEUPOGEN SOLN AGGRENOX CPMP 12HR anagrelide hcl capsule ARANESP SYRINGE ARANESP VIAL ARIXTRA SYRINGE cilostazol tablet COUMADIN TABLET COUMADIN VIAL CYKLOKAPRON AMPUL dipyridamole tablet EPOGEN VIAL HEPARIN SODIUM IN 0.45% NACL IV SOLN. HEPARIN SODIUM VIAL heparin sodium, porcine d5w iv soln. heparin sodium, porcine ns pf iv soln. LEUKINE VIAL LOVENOX SYRINGE LOVENOX VIAL NEULASTA SYRINGE pentoxifylline tablet sa PLAVIX TABLET PROCRIT VIAL ticlopidine hcl tablet warfarin sodium tablet PA and sinemet. A. There is in vitroand in vivo evidence that Micotilis a potent calcium channel antagonist with clinicalsigns moresimilarto dihydropyridine overdose, suchas nifedipine, as opposedto mixedion channelblockerslike verapamil. ~ Administration of intravenous calcium may provide benefit if patient is exhibiting rapid heart rate tachycardia ; or low blood pressure hypotension. Electrocution was proposed in the Gay 1890's as a "humane" alternative to hanging. Lethal injection was then adamantly opposed by the medical profession, concerned that defamation would accompany physician directed death. Thomas A. Edison, an opponent of capital punishment and inventor promoter of "safe" DC direct current ; , attempted to kill his major competitor Westinghouse by promoting AC alternating current ; for death-row denizens. Instead Westinghouse gained new respect and market share. But convicts did not always perish in "the chair." In 1903 the current surge left Clinton convict Frederick Van Wormer still breathing and twitching on the autopsy table. Sing-Sing Prison took over all NYS electrocutions in 1914. In 1995 capital punishment reverted back to Clinton, this time by lethal injection. Bellis M "Death and Money: The History of the Electric Chair" Inventors. About . : inventors.about library weekly aa102497 and hytrin. Evaluation of acceptability, tolerance and observance of a new calciumvitamine D combination Thomas J.L.; Meunier P.J. Hopital Edouard Herriot, Serv. de Rhumatologie Pathol. Oss., Place d'Arsonval, 69437 Lyon Cedex 3 France Rhumatologie France ; , 1996, 48 2 ; The aim of this trial was to assess in 190 patients randomized in two identical groups the tolerance and acceptability of a new calcium-vitamin D combination, OROCAL R ; Vitamine D3 chewable tablets containing calcium 500 mg and vitamine D3 400 IU ; in order to compare it with the same doses of calcium 1 g day ; and vitamin D 800 IU day ; , obtained by taking two SANDOCAL R ; 500 mg bags and two STEROGYL R ; drops day. After 10 weeks, patients were asked about the acceptability of the treatments and the occurrence of adverse effects. They had also to answer before and after treatment a guestionnaire listing 10 gastrointestinal symptoms. The observance and the acceptability have been better under OROCAL R ; Vitamine D3, with drop out 3 times less numerous under this combination than under the SANDOCAL R ; + STEROGYL R ; solution. The number of patients with a gastrointestinal symptom and the total number of these symptoms were statistically higher in the SANDOCAL R ; + STEROGYL R ; group, this association being more frequently responsible for flatulences, probably because the solution is effervescent. Many women who come to the Broussais Clinic are young and single with children; often they are in precarious social and financial situations. Sixty percent of clients have problems negotiating financial payments and getting the proper paperwork filled out so that they can receive the benefits to which they are entitled. Francine, the social worker at Broussais Clinic, assists clients experiencing socioeconomic problems. She helps the women maneuver through the complex French health care bureaucracy so that all who need care can receive it. At times, this involves finding creative solutions to clients' problems. Although providing this type of social assistance to women is not obligatory in French public-sector clinics, the staff believe that this service is important or many women would--for logistical, bureaucratic, or financial reasons--be denied the services they need and aripiprazole. Drug interactions rosiglitazone was shown to have no clinically relevant effect on the pharmacokinetics of nifedipine and oral contraceptives ethinylestradiol and norethindrone ; , which are predominantly metabolised by cyp3a rosiglitazone did not alter the metabolism of glyburide, metformin, acarbose, warfarin and digoxin when administered concomitantly.

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Erythromycin: Erythromycin, a CYP3A inhibitor, can inhibit the metabolism of nifedipine and increase the exposure to nifedipine during concomitant therapy. Blood pressure should be monitored and reduction of the dose of nifedipine considered. Antitubercular Drugs Rifampin: Pretreatment of healthy volunteers with 600 mg day rifampin p.o. decreased the exposure to oral nifedipine 20 g kg ; 13%. The exposure to intravenous nifedipine by the same rifampin treatment was decreased to 70%. Dose adjustment of nifedipine may be necessary if nifedipine is co-administered with rifampin. Rifapentine: Rifapentine, as an inducer of CYP3A4, can decrease the exposure to nifedipine. A dose adjustment of jifedipine when co-administered with rifapentine should be considered. Antiviral Drugs Amprenavir, atanazavir, delavirine, fosamprinavir, indinavir, nelfinavir and ritonavir, as CYP3A inhibitors, can inhibit the metabolism of nifedipinr and increase the exposure to nifedipine. Caution is warranted and clinical monitoring of patients recommended. CNS Drugs Nefazodone, a CYP3A inhibitor, can inhibit the metabolism of nifeeipine and increase the exposure to nifedipine during concomitant therapy. Blood pressure should be monitored and a reduction of the dose of nifedipine considered. Valproic acid may increase the exposure to nifedipine during concomitant therapy. Blood pressure should be monitored and a dose reduction of nifedipine considered. Phenytoin: Niffdipine is metabolized by CYP3A4. Co-administration of nifedipine 10 mg capsule and 60 mg nifedipine coat-core tablet with phenytoin, an inducer of CYP3A4, lowered the AUC and Cmax of nifedipine by approximately 70%. When using nifedipine with phenytoin, the clinical response to nifedipine should be monitored and its dose adjusted if necessary. Phenobarbitone and carbamazepine as inducers of CYP3A can decrease the exposure to nifedipine. Dose adjustment of nifedipine may be necessary if phenobarbitone, carbamazepine or phenytoin is co-administered. Antiemetic Drugs Dolasetron: In patients taking dolasetron by the oral or intravenous route and nifedipine, no effect was shown on the clearance of hydrodolasetron. Immunosuppressive Drugs Tacrolimus: Nifedipine has been shown to inhibit the metabolism of tacrolimus in vitro. Transplant patients on tacrolimus and nifedipine required from 26% to 38% smaller doses than patients not receiving nifedipine. Nifedipine can increase the exposure to tacrolimus. When nifedipine is co-administered with tacrolimus the blood concentrations of tacrolimus should be monitored and a reduction of the dose of tacrolimus considered. Sirolimus: A single 60 mg dose of nifedipine and a single 10 mg dose of sirolimus oral solution were administered to 24 healthy volunteers. Clinically significant pharmacokinetic drug interactions were not observed and quinapril.
Fluconazole * * Glibenclamide * Ciclosporin * Amiodarone Atorvastatin Fluvastatin Cimetidine Isoniazid Miconazole Voriconazole Ketoconazole Itraconazole Omeprazole ? ; Fluoxetine ? ; -- Fluoxetine Sertraline ? ; Paroxetine Fluvoxamine Sulfamethoxazole Clarithromycin Trimethoprim Erythromycin Zafirlukast Metronidazole Capecitabine Diltiazem Nifedipine Verapamil Quinine Nefazodone Grapefruit Juice Reboxetine.

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FC2.09.08 PRETERM LABOUR AND CALCIUM CHANNEL BLOCKERS Z. Ansari Chaharsoghy, Dept. OB GYN, Isfahan Medical School, Isfahan, Iran. Purpose: To determine efficacy of nifedipine for treatment of preterm labour. Method: In a case control study, 50 patients with preterm labour were separated into 2 groups and were treated with nifedipine and magnesium and aceon. In case of diuretics 'water pills' ; nifedipine adalat procardia ; and high blood pressure hypertension. Exercise tolerance was measured by a 6-min walking test at the screening visit and on days 8 and 23 of the respective treatment periods before inhalation of the study drug and perindopril. DISCUSSION Ulcers are among the most important complication of diabetes and they usually cause amputation of lower extremity. At the present there are several medical interventions to prevent and or treat chronic wounds including different dressings, use of antimicrobial agents and wound healing drugs 11 ; . Nifedipine, a calcium channel blocker, is being used for treatment of hypertensive venous leg ulcers and significantly improved skin wound healing process in patients received 10 mg Nifedipine three times a day 7 ; . Ward et al used Nifedipine to treat finger ulcers in scleroderma. Even reducing, parallel trade in their products. Companies that do not implement such strategies face a significant increase in parallel trade, as traders look to fill the gaps." After many years in which the industry's legal actions backfired in favor of parallel traders, the tide finally may have turned. "The European Court of Justice's final ruling upholding Bayer's supply management system for Adalat [nifedipine] in France and Spain provides further evidence of this sea change, " adds Haigh and sumycin and nifedipine. NIFEDIPINE SUSTAINED-RELEASE ; 20 MG TAB-CAP PO ; Price Tab-Cap 30 MG Supplier MEDS 1000 TAB-CAP 4.28 0.0043 TABLETS Supplier ACTION 2000 TAB-CAP 22.90 0.0114 TABLETS Supplier MISSION 100 TAB-CAP 1.45 TABLETS Supplier JMS 100 TAB-CAP 1.89 TABLETS Supplier IDA 100 TAB-CAP 2.03 TABLETS Supplier DURBIN 100 TAB-CAP 2.20 TABLETS Supplier IMRES 100 TAB-CAP 2.80 COATED TABLETS, BLISTER PACK Supplier Median Price Tab-Cap 0.0189 High Low Ratio 6.51 Buyer BDS 100 TAB-CAP 2.59 TABLETS Buyer MEMS 100 TAB-CAP 2.88 TABLETS Buyer OECS PPS 100 TAB-CAP 3.00 TABLETS Buyer Median Price Tab-Cap 0.0288 High Low Ratio 1.16 NITRAZEPAM 5 MG TAB-CAP PO ; Buyer BDS 500 TAB-CAP Buyer SAFRICA 250 TAB-CAP NITROFURANTOIN 100 MG TAB-CAP PO ; Supplier MEDS 1000 TAB-CAP Supplier MISSION 1000 TAB-CAP Supplier JMS 1000 TAB-CAP Supplier MEG 1000 TAB-CAP Supplier IMRES 1000 TAB-CAP Supplier ORBI 1000 TAB-CAP Supplier DURBIN 1000 TAB-CAP Price Tab-Cap 5 MG 6.33 0.0126 TABLETS 7.78 0.0311 TABLETS Buyer Median Price Tab-Cap 0.0219 High Low Ratio 2.47 Price Tab-Cap 0.2 G 3.56 0.0036 TABLETS 4.06 0.0041 TABLETS 5.49 0.0055 TABLETS 8.36 0.0084 TABLETS 9.52 0.0095 TABLETS 9.78 0.0098 TABLETS 13.96 0.0140 TABLETS Supplier Median Price Tab-Cap 0.0084 High Low Ratio 3.89 6.06 0.0061 TABLET 4.13 TABLETS 12.95 CAPSULES, BLISTER PACK 13.16 0.2632 MACROCRYSTALLINE CAPSULES Buyer Median Price Tab-Cap 0.0854 High Low Ratio 43.15 5.12 6.62 Price Ml 0.0427 0.2 G.
Yet the male response to the drug is not as pronounced as that seen in women and risedronate. The above recommendations are systematically developed statements to assist practitioner and patient decisions about appropriate health care for specific clinical circumstances. They should be used as an adjunct to sound clinical decision making.
Parison to placebo 29; Vocci, personal communication, 2000 ; . Methylphenidate MP ; is a stimulant and DA agonist primarily used in the treatment of childhood attention-deficit hyperactivity disorder. MP is a agonist with pharmacologic properties that include DA release, and it has similar levels of binding to the DAT as cocaine. Grabowski et al. 55 ; have reported that it does not increase cocaine use and retains patients better than placebo, but have not shown a reduction in cocaine use compared to placebo. Mazindol is a DA reuptake inhibitor that is without abuse liability and it has been suggested that it might antagonize the effects of cocaine as a treatment. A report on the effects of cocaine alone and in combination with mazindol at 1 or mg orally in cocaine abusing volunteers found that the combination significantly increased heart rate and blood pressure 56 ; . Mazindol did not alter the subjective effects of cocaine. One 12-week, double-blind, placebo-controlled clinical trial of mazindol 2 mg daily in cocainedependent subjects reported no difference from placebo 57 ; . Mazindol was also not well tolerated, with 16 of 33 patients dropping out, and the average length of treatment was 5 weeks. A similar trial in methadone maintained patients found limited efficacy for those patients who had been cocaine abstinent for at least 2 weeks before starting mazindol 58 ; . Nonspecific Anticraving Agents A number of other agents have been tested to reduce the desire or craving for cocaine. The rationales have broadly involved mechanisms such as sensitization and kindling as well as neurotransmitter systems that are indirectly affected by cocaine such as the opioid, excitatory amino acid glutamate, and GABAergic systems. For most of these approaches, outpatient clinical trials have been quite limited. Medications include GABA agents such as baclofen, opioid antagonists such as naltrexone, calcium channel blockers such as nifedipine, antikindling agents such as carbamazepine, and disulfiram. Finally, stress responses and the associated elevation of cortisol have been considered as potentially important in cocaine craving induction and as a therapeutic agent. However, a cocaine administration study showed no reduction in cocaine effects or self-administration with the cortisol synthesis inhibitor ketoconazole in spite of significant reductions in cortisol levels 59 ; . Carbamazepine CBZ ; is an anticonvulsant medication hypothesized to have potential as a treatment for cocaine craving and abuse because of its ability to block cocaineinduced ``kindling'' in rodents. A double-blind, placebocontrolled, crossover study of the interaction of 400 mg of CBZ daily for 5 days with cocaine found no effects on subjective response to cocaine 60 ; . A double-blind, placebo-controlled study in outpatients included a 20-day, controlled, fixed-dose CBZ 200 mg or 400 mg or placebo. 1. All of the following antiepileptic agents are considered weak enzyme inducers, EXCEPT: A. Felbamate B. Topiramate C. Gabapentin D. Oxcarbazepine 2. All of the following antiepileptic agents are considered strong enzyme inducers, EXCEPT: A. Carbamazepine B. Valproate C. Phenobarbital D. Phenytoin 3. All of the following are considered non-inducing antiepileptic agents, EXCEPT: A. Levetiracetam B. Pregabalin C. Valproate D. Tiagabine E. Topiramate 4. Enzyme inhibition may result in: A. Higher plasma concentrations of substrate drugs B. Prolonged half-life of substrate drugs C. Increased potential for adverse events D. All of the above 5. Enzyme induction may result in: A. Increased metabolism of substrate drugs B. Lower plasma concentrations of substrate drugs C. Possible reduction in therapeutic efficacy of substrate drugs D. All of the above 6. If a patient is receiving concomitant oxcarbazepine and nifedipine treatment, the patient should be monitored for: A. Decreased oxcarbazepine concentrations B. Increased oxcarbazepine concentrations C. Decreased nifedipine concentrations D. Increased nifedipine concentrations 7. Which of the following antiepileptic agents would NOT interact with oral contraceptives taken by a female patient? A. Valproate B. Oxcarbazepine C. Felbamate D. Topiramate 8. Which statement BEST describes the relationship between carbamazepine phenobarbital phenytoin and enzyme induction? A. Carbamazepine is a more potent inducer than phenobarbital and phenytoin B. Phenobarbital is a more potent inducer than carbamazepine and phenytoin C. Phenytoin is a more potent inducer than carbamazepine and phenobarbital D. The actual dosage may play a more important role than which agent is used 9. The time course of enzyme induction is dependent on: A. The half-life of the inducing agent B. The half-life of the induced enzyme C. The half-life of the substrate drug D. A and B E. A and C. Twelve Satellite Symposia organized by medical industries will be held during APDW 2005. Lunch and dinner will be offered to the participants of the Satellite Symposia, because nifedipine extended release tablets. BANGKOK DRUG BIOLAB BIOMEDIS FARMALINE GREATER PHARM M&H MANUFACTURING MILLIMED NEW LIFE PHARMA NIDA PHARMA OLAN OSOTH INTER LABORA OSOTH INTER LABORA PHARMAHOF POLIPHARM PONDS CHEMICAL PROGRESS MED. RX.CO-PH SANOFI AVENTIS SIAM BHAESAJ CO SRIPRASIT PHARMA T.MAN PHARMA T.O.CHEMICAL UTOPIAN V.S. PHARM CHAROEN BHAESAJ GENERAL DRUG HOUSE POLIPHARM THE MEDIC PHARM POLIPHARM T.P.DRUG LAB THE MEDIC PHARM SANOFI AVENTIS THE MEDIC PHARM UNISON T.P.DRUG LAB R.X COMPANY THAI NAKORN PATANA BIOLAB CHIESI GLAXOSMITHKLINE RIKER LAB AUST PTY ORION PHARM CIPLA LAB ALDO-UNION 133 and reminyl.
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