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However, in contrast to the results of the challenge experiments with SupT cells Fig. 4A and B ; , the combination of PTX and Go-6976 re sulted in a significant improvement in protection compared to cells treated with Go-6976 alone. This pattern of inhibition of virus replication by the combination of NF- B inhibitors correlated well with their effects on HIV-1 LTR-regulated gene expression Fig. 3B ; . Prolonged inhibition of HIV-1 infection in stably transfected CD4 SupT cells and in transduced PBMCs and CD4 mononuclear cells expressing Anti-Tat sFv intrabodies in the presence of NF- B inhibitors. Since treatment of cells with the combination of NF- B and Tat inhibitors results in cooperative down-regulation of Tat-mediated activation of the HIV-1 promoter Fig. 3A and references 6 and 7 ; , these observations suggested that a combined genetic and pharmacologic treatment strategy might result in a more durable inhibition of HIV-1 replication. 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